Disrupted Membrane Homeostasis and Accumulation of Ubiquitinated Proteins in a Mouse Model of Infantile Neuroaxonal Dystrophy Caused by PLA2G6 Mutations-Reference-Cited by-同舟云学术

Disrupted Membrane Homeostasis and Accumulation of Ubiquitinated Proteins in a Mouse Model of Infantile Neuroaxonal Dystrophy Caused by PLA2G6 Mutations

Published:2008-02 Issue:2 Volume:172 Page:406-416
ISSN:0002-9440
Container-title:The American Journal of Pathology
language:en
Short-container-title:The American Journal of Pathology

Author:

Malik Ibrahim,Turk John,Mancuso David J.,Montier Laura,Wohltmann Mary,Wozniak David F.,Schmidt Robert E.,Gross Richard W.,Kotzbauer Paul T.

Publisher

Elsevier BV

Subject

Pathology and Forensic Medicine

Reference42 articles.

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3. Histological and ultrastructural features of dystrophic isocortical axons in infantile neuroaxonal dystrophy (Seitelberger's disease);de Leon;Acta Neuropathol (Berl),1985

4. Infantile neuroaxonal dystrophy (Seitelberger's disease). A light and ultrastructural study;Yagishita;Acta Neuropathol (Berl),1975

5. An analysis of the ultrastructural findings in infantile neuroaxonal dystrophy (Seitelberger's disease);Liu;Acta Neuropathol (Berl),1974

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