Possible Origin of Rat Testicular Atrophy Induced by Di-n-Butyl Phthalate: Changes in the Activities of Some Enzymes during Rat Testis Perfusion under a Hypoxic Condition and with Mono-n-Butyl Phthalate.-Reference-Cited by-同舟云学术

Possible Origin of Rat Testicular Atrophy Induced by Di-n-Butyl Phthalate: Changes in the Activities of Some Enzymes during Rat Testis Perfusion under a Hypoxic Condition and with Mono-n-Butyl Phthalate.

Published:2002 Issue:6 Volume:48 Page:503-513
ISSN:1344-9702
Container-title:Journal of Health Science
language:en
Short-container-title:JOURNAL OF HEALTH SCIENCE

Author:

Watanabe Nobuo¹,Shimizu Makiko¹,Matsumoto Yoshiaki¹,Fukuoka Masamichi¹

Affiliation:

1. Department of Clinical Pharmacology and Toxicology, Showa Pharmaceutical University

Publisher

Pharmaceutical Society of Japan

Subject

Health, Toxicology and Mutagenesis,Toxicology

Link

http://www.jstage.jst.go.jp/article/jhs/48/6/48_6_503/_pdf

Reference38 articles.

1. The clinical effects of plasticizers, antioxidants, and other contaminants in medical polyvinylchloride tubing during respiratory and non-respiratory exposure

2. Potential Hazards of Exposure to Di-(2-Ethylhexyl)-Phthalate in Babies

3. Hormones and testis development and the possible adverse effects of environmental chemicals

4. Effects of di-n-butyl phthalate (DBP) on male reproductive development in the rat: implications for human risk assessment

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1. Proteome profiling of mouse embryonic stem cells to define markers for cell differentiation and embryotoxicity;Reproductive Toxicology;2010-09

2. Testicular Toxicity of Mono-n-Butyl Phthalate and Related Phthalates on Bound Iron in Rat Red Blood Cells.;Journal of Health Science;2002