Ginsenoside Re protects against kainate-induced neurotoxicity in mice by attenuating mitochondrial dysfunction through activation of the signal transducers and activators of transcription 3 signaling

Author:

Nguyen Yen Nhi Doan1,Jeong Ji Hoon2,Sharma Naveen12,Tran Ngoc Kim Cuong1,Tran Hoang-Yen Phi3,Dang Duy-Khanh4,Park Jung Hoon1,Byun Jae Kyung5,Ko Sung Kwon6,Nah Seung-Yeol7,Kim Hyoung-Chun1,Shin Eun-Joo1

Affiliation:

1. Neuropsychopharmacology and Toxicology Program, College of Pharmacy, Kangwon National University, Chunchon, Republic of Korea

2. Department of Global Innovative Drugs, Graduate School of Chung-Ang University, College of Medicine, Chung-Ang University, Seoul, Republic of Korea

3. Department of Physical Chemistry, University of Medicine and Pharmacy at Ho Chi Minh City, Viet Nam, Ho Chi Minh City

4. Department of Pharmacy, Can Tho University of Medicine and Pharmacy, Can Tho City, Viet Nam, Ho Chi Minh City

5. Korea Society of Forest Environmental Research, Namyangju, Republic of Korea

6. Department of Oriental Medical Food & Nutrition, Semyung University, Jecheon, Republic of Korea

7. Ginsentology Research Laboratory and Department of Physiology, College of Veterinary Medicine, Konkuk University, Seoul, Republic of Korea

Funder

R&D Program for Forest Science Technology

Korea Forest Service

Basic Science Research Program

National Research Foundation of Korea

Ministry of Science and ICT

Republic of Korea

Korea Basic Science Institute

Ministry of Education

Republic of Korea and Ministry of Science and ICT, South Korea

Publisher

Informa UK Limited

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