Testicular Germ-Cell Apoptosis in Stressed Rats Following Combined Exposure to Pyridostigmine Bromide, N , N -Diethyl m -Toluamide (Deet), and Permethrin
Author:
Publisher
Informa UK Limited
Subject
Health, Toxicology and Mutagenesis,Toxicology
Link
http://www.tandfonline.com/doi/pdf/10.1080/15287390306463
Reference52 articles.
1. Subchronic Dermal Application of N,N-Diethyl m-Toluamide (DEET) and Permethrin to Adult Rats, Alone or in Combination, Causes Diffuse Neuronal Cell Death and Cytoskeletal Abnormalities in the Cerebral Cortex and the Hippocampus, and Purkinje Neuron Loss in the Cerebellum
2. Increased Neurotoxicity Following Concurrent Exposure to Pyridostigmine Bromide, DEET, and Chlorpyrifos
3. NEUROTOXICITY RESULTING FROM COEXPOSURE TO PYRIDOSTIGMINE BROMIDE, DEET, AND PERMETHRIN: IMPLICATIONS OF GULF WAR CHEMICAL EXPOSURES
4. EFFECTS OF DAILY DERMAL APPLICATION OF DEET AND PERMETHRIN, ALONE AND IN COMBINATION, ON SENSORIMOTOR PERFORMANCE, BLOOD-BRAIN BARRIER, AND BLOOD-TESTIS BARRIER IN RATS
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2. Altered gene expression linked to germline dysfunction following exposure of Caenorhabditis elegans to DEET;iScience;2024-01
3. Interaction between Pyridostigmine Bromide and Oxidative Stress;Medical Toxicology;2021-02-03
4. The Val16Ala-SOD2 polymorphism affects cyto-genotoxicity of pyridostigmine bromide on human peripheral blood mononuclear cells;Toxicology in Vitro;2019-10
5. Testicular Cancer: Genes, Environment, Hormones;Frontiers in Endocrinology;2019-07-02
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