NCBP3/SNHG6 inhibits GBX2 transcription in a histone modification manner to facilitate the malignant biological behaviour of glioma cells-Reference-Cited by-同舟云学术

NCBP3/SNHG6 inhibits GBX2 transcription in a histone modification manner to facilitate the malignant biological behaviour of glioma cells

Published:2020-07-26 Issue:1 Volume:18 Page:47-63
ISSN:1547-6286
Container-title:RNA Biology
language:en
Short-container-title:RNA Biology

Author:

Li Xiwen¹²³,Zhang Fangfang¹²³,Ma Jun¹²³,Ruan Xuelei¹²³,Liu Xiaobai⁴⁵⁶,Zheng Jian⁴⁵⁶,Liu Yunhui⁴⁵⁶,Cao Shuo¹²³,Shen Shuyuan¹²³,Shao Lianqi¹²³,Cai Heng⁴⁵⁶,Li Zhen⁴⁵⁶,Xue Yixue¹²³

Affiliation:

1. Department of Neurobiology, School of Life Sciences, China Medical University, Shenyang, China

2. Key Laboratory of Cell Biology, Ministry of Public Health of China, China Medical University, Shenyang, China

3. Key Laboratory of Medical Cell Biology, Ministry of Education of China, China Medical University, Shenyang, China

4. Department of Neurosurgery, Shengjing Hospital of China Medical University, Shenyang, China

5. Liaoning Clinical Medical Research Center in Nervous System Disease, Shenyang, China

6. Key Laboratory of Neuro-oncology in Liaoning Province, Shenyang, China

Funder

National Natural Science Foundation of China

China Postdoctoral Science Foundation

Liaoning Science and Technology Plan Project

Project of Key Laboratory of Neuro-oncology in Liaoning Province

Publisher

Informa UK Limited

Subject

Cell Biology,Molecular Biology

Link

https://www.tandfonline.com/doi/pdf/10.1080/15476286.2020.1790140

Reference85 articles.

1. The epidemiology of glioma in adults: a "state of the science" review

2. Exciting New Advances in Neuro-Oncology: The Avenue to a Cure for Malignant Glioma

3. Dominant-negative inhibition of the Axl receptor tyrosine kinase suppresses brain tumor cell growth and invasion and prolongs survival

4. EIF4A3-induced circular RNA MMP9 (circMMP9) acts as a sponge of miR-124 and promotes glioblastoma multiforme cell tumorigenesis

5. Honokiol Eliminates Glioma/Glioblastoma Stem Cell-Like Cells Via JAK-STAT3 Signaling and Inhibits Tumor Progression by Targeting Epidermal Growth Factor Receptor

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