Inhibition of Src but not Syk causes weak reversal of GPVI-mediated platelet aggregation measured by light transmission aggregometry-Reference-Cited by-同舟云学术

Inhibition of Src but not Syk causes weak reversal of GPVI-mediated platelet aggregation measured by light transmission aggregometry

Published:2022-05-09 Issue:8 Volume:33 Page:1293-1300
ISSN:0953-7104
Container-title:Platelets
language:en
Short-container-title:Platelets

Author:

Cheung Hilaire Yam Fung¹²³^ORCID,Moran Luis A.¹⁴^ORCID,Sickmann Albert²^ORCID,Heemskerk Johan W.M.³^ORCID,Garcia Ángel⁴^ORCID,Watson Steve P.¹^ORCID

Affiliation:

1. Institute of Cardiovascular Sciences, College of Medical and Dental Sciences, University of Birmingham, Birmingham, UK

2. Department of Bioanalytics, Leibniz-Institut für Analytische Wissenschaften-ISAS-e.V, Dortmund, Germany

3. Department of Biochemistry, Cardiovascular Research Institute Maastricht (CARIM), Maastricht University, Maastricht, The Netherlands

4. Center for Research in Molecular Medicine and Chronic Diseases (CIMUS), Universidad de Santiago de Compostela, and Instituto de Investigación Sanitaria (IDIS), Santiago de Compostela, Spain

Funder

British Heart Foundation

European Union’s Horizon 2020 Marie Skłodowska-Curie Actions

Publisher

Informa UK Limited

Subject

Hematology,General Medicine

Link

https://www.tandfonline.com/doi/pdf/10.1080/09537104.2022.2069235

Reference18 articles.

1. GPVI and CLEC-2 in hemostasis and vascular integrity

2. Pharmacological Blockade of Glycoprotein VI Promotes Thrombus Disaggregation in the Absence of Thrombin

3. Critical role for Syk in responses to vascular injury

4. Immobilized fibrinogen activates human platelets through glycoprotein VI

5. Ability of anti-glycoprotein IIb/IIIa agents to dissolve platelet thrombi formed on a collagen surface under blood flow conditions

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