Endomembrane damage sensing by V-ATPase recruits ATG16L1 for LC3 lipidation <i>in situ</i>-Reference-Cited by-同舟云学术

Endomembrane damage sensing by V-ATPase recruits ATG16L1 for LC3 lipidation in situ

Published:2022-04-12 Issue:11 Volume:18 Page:2751-2753
ISSN:1554-8627
Container-title:Autophagy
language:en
Short-container-title:Autophagy

Author:

Xu Yue¹,Ding Jingjin²^ORCID

Affiliation:

1. Key Laboratory of Cell Differentiation and Apoptosis of Chinese Ministry of Education, Department of Pathophysiology, Shanghai Jiao Tong University School of Medicine, Shanghai, China

2. National Laboratory of Biomacromolecules, CAS Center for Excellence in Biomacromolecules, Institute of Biophysics, Chinese Academy of Sciences, Beijing, China

Funder

Chinese Academy of Sciences

Excellent Young Scholar Program

National Natural Science Foundation of China

National Key Research and Development Program of China

Youth Innovation Promotion Association CAS

Publisher

Informa UK Limited

Subject

Cell Biology,Molecular Biology

Link

https://www.tandfonline.com/doi/pdf/10.1080/15548627.2022.2062889

Reference1 articles.

1. ARF GTPases activate Salmonella effector SopF to ADP-ribosylate host V-ATPase and inhibit endomembrane damage-induced autophagy

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