Chronic HMGCR/HMG-CoA reductase inhibitor treatment contributes to dysglycemia by upregulating hepatic gluconeogenesis through autophagy induction

Author:

Wang Hye Jin123,Park Jae Yeo45,Kwon Obin36,Choe Eun Yeong12,Kim Chul Hoon36,Hur Kyu Yeon7,Lee Myung-Shik78,Yun Mijin9,Cha Bong Soo123,Kim Young-Bum10,Lee Hyangkyu45,Kang Eun Seok123

Affiliation:

1. Division of Endocrinology and Metabolism; Department of Internal Medicine; Yonsei University College of Medicine; Seoul, Korea

2. Institute of Endocrine Research; Yonsei University College of Medicine; Seoul, Korea

3. Brain Korea 21 PLUS Project for Medical Science; Yonsei University College of Medicine; Seoul, Korea

4. Department of Clinical Nursing Science; Yonsei University College of Nursing; Seoul, Korea

5. Nursing Policy and Research Institute; Biobehavioral Research Center; Yonsei University; Seoul, Korea

6. Department of Pharmacology; Yonsei University College of Medicine; Seoul, Korea

7. Department of Medicine; Samsung Medical Center; Sungkyunkwan University School of Medicine; Seoul, Korea

8. Samsung Advanced Institute for Health Sciences and Technology; Sungkyunkwan University School of Medicine; Seoul, Korea

9. Department of Nuclear Medicine; Yonsei University College of Medicine; Seoul, Korea

10. Division of Endocrinology, Diabetes, and Metabolism; Department of Medicine; Beth Israel Deaconess Medical Center and Harvard Medical School; Boston, MA USA

Publisher

Informa UK Limited

Subject

Cell Biology,Molecular Biology

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