Excessive fibrinolysis in AL-amyloidosis is induced by urokinae-type plasminogen activator from bone marrow plasma cells
Author:
Publisher
Informa UK Limited
Subject
Internal Medicine
Link
http://www.tandfonline.com/doi/pdf/10.1080/13506120902879269
Reference17 articles.
1. Hemostatic Dysfunction in Paraproteinemias and Amyloidosis
2. Direct evidence for systemic fibrinogenolysis in patients with acquired α2-plasmin inhibitor deficiency
3. Increased plasmin-α2-antiplasmin levels indicate activation of the fibrinolytic system in systemic amyloidoses
4. Elevated urokinase-type plasminogen activator level and bleeding in amyloidosis: Case report and literature review
5. Excessive Fibrinolysis in Amyloidosis Associated with Elevated Plasma Single-Chain Urokinase
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4. Hemostasis disorders in patients with systemic AL-amyloidosis;Terapevticheskii arkhiv;2023-11-03
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