Apremilast reversed carfilzomib-induced cardiotoxicity through inhibition of oxidative stress, NF-κB and MAPK signaling in rats-Reference-Cited by-同舟云学术

Apremilast reversed carfilzomib-induced cardiotoxicity through inhibition of oxidative stress, NF-κB and MAPK signaling in rats

Published:2016-10-27 Issue:9 Volume:26 Page:700-708
ISSN:1537-6516
Container-title:Toxicology Mechanisms and Methods
language:en
Short-container-title:Toxicology Mechanisms and Methods

Author:

Imam Faisal¹,Al-Harbi Naif O.¹,Al-Harbi Mohammad Matar¹,Ansari Mushtaq Ahmad¹,Almutairi Mashal M¹,Alshammari Musaad¹,Almukhlafi Talal Saad²,Ansari Mohd Nazam²,Aljerian Khaldoon³,Ahmad Sheikh Fayaz¹

Affiliation:

1. Department of Pharmacology and Toxicology, College of Pharmacy, King Saud University, Riyadh, Saudi Arabia;

2. Department of Pharmacology, College of Pharmacy, Prince Sattam Bin Abdulaziz University, Al-Kharj, Saudi Arabia;

3. King Khalid University Hospital, College of Medicine, King Saud University, Forensic Medicine and Toxicology Unit, Riyadh, Saudi Arabia

Publisher

Informa UK Limited

Subject

Health, Toxicology and Mutagenesis,Toxicology

Link

https://www.tandfonline.com/doi/pdf/10.1080/15376516.2016.1236425

Reference74 articles.

1. Classification and natural history of primary myocardial disease

2. Carfilzomib-induced cardiotoxicity mitigated by dexrazoxane through inhibition of hypertrophic gene expression and oxidative stress in rats

3. Riboflavin attenuates lipopolysaccharide-induced lung injury in rats

4. Phosphodiesterase III inhibitors for heart failure

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