Mitochondrial dysfunction in Alzheimer’s disease - a proteomics perspective-Reference-Cited by-同舟云学术

Mitochondrial dysfunction in Alzheimer’s disease - a proteomics perspective

Published:2021-04-03 Issue:4 Volume:18 Page:295-304
ISSN:1478-9450
Container-title:Expert Review of Proteomics
language:en
Short-container-title:Expert Review of Proteomics

Author:

Abyadeh Morteza¹^ORCID,Gupta Vivek²,Chitranshi Nitin²,Gupta Veer³,Wu Yunqi⁴,Saks Danit²,Wander Wall Roshana²,Fitzhenry Matthew J.⁴,Basavarajappa Devaraj²,You Yuyi²,Salekdeh Ghasem H⁵,Haynes Paul a⁵,Graham Stuart L²,Mirzaei Mehdi²

Affiliation:

1. Cell Science Research Center, Department of Molecular Systems Biology, Royan Institute for Stem Cell Biology and Technology, ACECR, Tehran Iran

2. Department of Clinical Medicine, Macquarie University, Macquarie Park, NSW, Australia

3. School of Medicine, Deakin University, VIC, Australia

4. Australian Proteome Analysis Facility, Macquarie University, Macquarie Park, NSW Australia

5. Department of Molecular Sciences, Macquarie University, Macquarie Park, NSW, Australia

Funder

the Ophthalmic Research Institute of Australia

Publisher

Informa UK Limited

Subject

Molecular Biology,Biochemistry

Link

https://www.tandfonline.com/doi/pdf/10.1080/14789450.2021.1918550

Reference107 articles.

1. Amyloid-β signals through tau to drive ectopic neuronal cell cycle re-entry in Alzheimer's disease

2. The neuropathological diagnosis of Alzheimer’s disease

3. Global, regional, and national burden of Alzheimer's disease and other dementias, 1990–2016: a systematic analysis for the Global Burden of Disease Study 2016

4. Trends in the incidence and prevalence of Alzheimer's disease, dementia, and cognitive impairment in the United States

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