Loss of Ephaptic Contacts in the Murine Thalamus during Osmotic Demyelination Syndrome-Reference-Cited by-同舟云学术

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Loss of Ephaptic Contacts in the Murine Thalamus during Osmotic Demyelination Syndrome

Published:2023-07-21 Issue:5 Volume:47 Page:398-423
ISSN:0191-3123
Container-title:Ultrastructural Pathology
language:en
Short-container-title:Ultrastructural Pathology

Author:

Gilloteaux Jacques¹²,De Swert Kathleen¹,Suain Valérie³,Brion Jean-Pierre³,Nicaise Charles¹

Affiliation:

1. Department of Medicine, URPHyM, NARILIS, Université de Namur, Namur, Belgium

2. Department of Anatomical Sciences, St George’s University School of Medicine, Newcastle Upon Tyne, UK

3. Laboratory of Histology, Neuroanatomy and Neuropathology, Faculty of Medicine, Université Libre de Bruxelles, Brussels, Belgium

Funder

none

“Fonds de la Recherche Scientifique Médicale” , Brussels, Belgium

Publisher

Informa UK Limited

Subject

Structural Biology,Pathology and Forensic Medicine

Link

https://www.tandfonline.com/doi/pdf/10.1080/01913123.2023.2232452

Reference158 articles.

1. Regional oligodendrocytopathy and astrocytopathy precede myelin loss and blood-brain barrier disruption in a murine model of osmotic demyelination syndrome

2. Ultrastructural Analysis of Thalamus Damages in a Mouse Model of Osmotic-Induced Demyelination

3. The osmotic demyelination syndrome: the resilience of thalamic neurons is verified with transmission electron microscopy

4. Blood-brain barrier permeability towards small and large tracers in a mouse model of osmotic demyelination syndrome

5. A primary cilium in oligodendrocytes: a fine structure signal of repairs in thalamic Osmotic Demyelination Syndrome (ODS)

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Thalamic Neuron Resilience during Osmotic Demyelination Syndrome (ODS) Is Revealed by Primary Cilium Outgrowth and ADP-ribosylation factor-like protein 13B Labeling in Axon Initial Segment;International Journal of Molecular Sciences;2023-11-17

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