GRK2 and GRK5 as therapeutic targets and their role in maladaptive and pathological cardiac hypertrophy
Author:
Affiliation:
1. Center for Translational Medicine, Department of Pharmacology, Lewis Katz School of Medicine, Temple University, Philadelphia, PA, USA
Funder
National Institute of Health grants
and an American Heart Association grant
Publisher
Informa UK Limited
Subject
Clinical Biochemistry,Drug Discovery,Pharmacology,Molecular Medicine
Link
https://www.tandfonline.com/doi/pdf/10.1080/14728222.2019.1575363
Reference93 articles.
1. Prognostic Implications of Echocardiographically Determined Left Ventricular Mass in the Framingham Heart Study
2. The epidemiology of heart failure: The Framingham Study
3. Mechanisms of Exercise-Induced Cardioprotection
4. MAPK-Activated Protein Kinase-2 in Cardiac Hypertrophy and Cyclooxygenase-2 Regulation in Heart
5. Enhanced Cardiac Akt/Protein Kinase B Signaling Contributes to Pathological Cardiac Hypertrophy in Part by Impairing Mitochondrial Function via Transcriptional Repression of Mitochondrion-Targeted Nuclear Genes
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