The RORγt-CCR6-CCL20 axis augments Th17 cells invasion into the synovia of rheumatoid arthritis patients-Reference-Cited by-同舟云学术

The RORγt-CCR6-CCL20 axis augments Th17 cells invasion into the synovia of rheumatoid arthritis patients

Published:2018-01-22 Issue:5 Volume:28 Page:814-825
ISSN:1439-7595
Container-title:Modern Rheumatology
language:en
Short-container-title:Modern Rheumatology

Author:

Kaneko Shunta¹,Kondo Yuya¹,Yokosawa Masahiro¹,Furuyama Kotona¹,Segawa Seiji¹,Tsuboi Hiroto¹,Kanamori Akihiro²,Matsumoto Isao¹,Yamazaki Masashi²,Sumida Takayuki¹

Affiliation:

1. Department of Internal Medicine, Faculty of Medicine, University of Tsukuba, Tsukuba, Japan;

2. Department of Orthopedic Surgery, Faculty of Medicine, University of Tsukuba, Tsukuba, Japan

Funder

Ministry of Health, Labor and Welfare, Japan

JSPS KAKENHI

Publisher

Oxford University Press (OUP)

Subject

Rheumatology

Link

https://www.tandfonline.com/doi/pdf/10.1080/14397595.2017.1416923

Reference48 articles.

1. The Pathogenesis of Rheumatoid Arthritis

2. Helper T cell diversity and plasticity

3. Treatment with a neutralizing anti-murine interleukin-17 antibody after the onset of collagen-induced arthritis reduces joint inflammation, cartilage destruction, and bone erosion

4. Interleukin 17 contributes to the chronicity of inflammatory diseases such as rheumatoid arthritis

5. Inflammatory Mediators Alter Interleukin-17 Receptor, Interleukin-12 and -23 Expression in Human Osteoarthritic and Rheumatoid Arthritis Synovial Fibroblasts: Immunomodulation by Vasoactive Intestinal Peptide

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