Increased expression of miR-22 corresponds to the high-risk subtypes of myelodysplastic syndromes and lower OS rate
Author:
Affiliation:
1. Center for Tumor Diagnosis & Therapy, Jinshan Hospital of Fudan University, Shanghai, China
Publisher
Informa UK Limited
Subject
Cancer Research,Oncology,Hematology
Link
https://www.tandfonline.com/doi/pdf/10.1080/10428194.2020.1734591
Reference10 articles.
1. The Oncogenic MicroRNA miR-22 Targets the TET2 Tumor Suppressor to Promote Hematopoietic Stem Cell Self-Renewal and Transformation
2. TET2 expression level and 5-hydroxymethylcytosine are decreased in refractory cytopenia of childhood
3. miR-22 has a potent anti-tumour role with therapeutic potential in acute myeloid leukaemia
4. The PU.1-Modulated MicroRNA-22 Is a Regulator of Monocyte/Macrophage Differentiation and Acute Myeloid Leukemia
5. microRNA-22 promotes megakaryocyte differentiation through repression of its target, GFI1
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1. MicroRNA dysregulation in myelodysplastic syndromes: implications for diagnosis, prognosis, and therapeutic response;Frontiers in Oncology;2024-08-02
2. The Role of Non-Coding RNAs in Myelodysplastic Neoplasms;Cancers;2023-09-30
3. Extracellular Vesicles and MicroRNA in Myelodysplastic Syndromes;Cells;2023-02-19
4. Credit Risk Prediction Based on DenseNet-BC of Fusion Focal Loss and Static restart SGD;2021 IEEE SmartWorld, Ubiquitous Intelligence & Computing, Advanced & Trusted Computing, Scalable Computing & Communications, Internet of People and Smart City Innovation (SmartWorld/SCALCOM/UIC/ATC/IOP/SCI);2021-10
5. MicroRNAs in the Myelodysplastic Syndrome;Acta Naturae;2021-07-27
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