Disruption of CDK-resistant chromatin association by pRB causes DNA damage, mitotic errors, and reduces Condensin II recruitment
Author:
Affiliation:
1. London Regional Cancer Program, London, Ontario, Canada
2. Department of Biochemistry, Western University, London, Ontario, Canada
3. Children's Health Research Institute, London, Ontario, Canada
Publisher
Informa UK Limited
Subject
Cell Biology,Developmental Biology,Molecular Biology
Link
https://www.tandfonline.com/doi/pdf/10.1080/15384101.2017.1338984
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1. Active Localization of the Retinoblastoma Protein in Chromatin and Its Response to S Phase DNA Damage
2. Nucleotide Deficiency Promotes Genomic Instability in Early Stages of Cancer Development
3. The N Terminus of the Retinoblastoma Protein Inhibits DNA Replication via a Bipartite Mechanism Disrupted in Partially Penetrant Retinoblastomas
4. Distinct Action of the Retinoblastoma Pathway on the DNA Replication Machinery Defines Specific Roles for Cyclin-Dependent Kinase Complexes in Prereplication Complex Assembly and S-Phase Progression
5. G1 Cyclin/Cyclin-dependent Kinase-coordinated Phosphorylation of Endogenous Pocket Proteins Differentially Regulates Their Interactions with E2F4 and E2F1 and Gene Expression
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