Ablation of TRPV1-positive nerves exacerbates salt-induced hypertension and tissue injury in rats after renal ischemia-reperfusion via infiltration of macrophages-Reference-Cited by-同舟云学术

Ablation of TRPV1-positive nerves exacerbates salt-induced hypertension and tissue injury in rats after renal ischemia-reperfusion via infiltration of macrophages

Published:2020-12-16 Issue:3 Volume:43 Page:254-262
ISSN:1064-1963
Container-title:Clinical and Experimental Hypertension
language:en
Short-container-title:Clinical and Experimental Hypertension

Author:

Yu Shuang-Quan¹,Ma Shuangtao¹,Wang Donna H.¹²³

Affiliation:

1. Division of Nanomedicine and Molecular Intervention, Department of Medicine, Michigan State University, East Lansing, MI, USA

2. Neuroscience Program, Michigan State University, East Lansing, MI, USA

3. Cell and Molecular Biology Program, Michigan State University, East Lansing, MI, USA

Funder

National Institutes of Health

Publisher

Informa UK Limited

Subject

General Medicine,Physiology,Internal Medicine

Link

https://www.tandfonline.com/doi/pdf/10.1080/10641963.2020.1860078

Reference31 articles.

1. The capsaicin receptor: a heat-activated ion channel in the pain pathway

2. Rapid Communication: Salt-Sensitive Hypertension Induced by Sensory Denervation

3. Antihypertensive Mechanisms Underlying a Novel Salt-Sensitive Hypertensive Model Induced by Sensory Denervation

4. Degeneration of Capsaicin-Sensitive Sensory Nerves Leads to Increased Salt Sensitivity Through Enhancement of Sympathoexcitatory Response

5. Deletion of Transient Receptor Potential Vanilloid Type 1 Receptors Exaggerates Renal Damage in Deoxycorticosterone Acetate-Salt Hypertension

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