Hippocampal NG2+ pericytes in chronically stressed rats and depressed patients: a quantitative study-Reference-Cited by-同舟云学术

Hippocampal NG2+ pericytes in chronically stressed rats and depressed patients: a quantitative study

Published:2020-06-24 Issue:3 Volume:24 Page:353-358
ISSN:1025-3890
Container-title:Stress
language:en
Short-container-title:Stress

Author:

Treccani Giulia¹²³⁴,Schlegelmilch Anna-Lena²,Schultz Nina⁴,Herzog David P.²,Bessa Joao M.⁵⁶,Sotiropoulos Ioannis⁵⁶,Müller Marianne B.²³,Wennström Malin⁴

Affiliation:

1. Translational Neuropsychiatry Unit, Department of Clinical Medicine, Aarhus University, Aarhus, Denmark

2. Department of Psychiatry and Psychotherapy, University Medical Center of the Johannes Gutenberg-University Mainz, Mainz, Germany

3. Institute for Microscopic Anatomy and Neurobiology, University Medical Center of the Johannes Gutenberg-University Mainz, Mainz, Germany

4. Department of Clinical Sciences Malmö, Clinical Memory Research Unit, Lund University, Malmö, Sweden

5. Life and Health Sciences Research Institute (ICVS), Medical School, University of Minho, Braga, Portugal

6. ICVS/3B’s – PT Government Associate Laboratory, Braga, Guimarães, Portugal

Funder

Danish Council for Independent Research

Swedish Research Council

Publisher

Informa UK Limited

Subject

Behavioral Neuroscience,Psychiatry and Mental health,Endocrine and Autonomic Systems,Neuropsychology and Physiological Psychology,Physiology

Link

https://www.tandfonline.com/doi/pdf/10.1080/10253890.2020.1781083

Reference28 articles.

1. The role of pericytes in blood-vessel formation and maintenance

2. The mood-improving actions of antidepressants do not depend on neurogenesis but are associated with neuronal remodeling

3. Markers of central inflammation in major depressive disorder: A systematic review and meta-analysis of studies examining cerebrospinal fluid, positron emission tomography and post-mortem brain tissue

4. Early Loss of Pericytes and Perivascular Stromal Cell-Induced Scar Formation after Stroke

5. Putative Inflammatory Sensitive Mechanisms Underlying Risk or Resilience to Social Stress

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