Distribution and progression of cerebral amyloid angiopathy in early-onset V30M (p.V50M) hereditary ATTR amyloidosis-Reference-Cited by-同舟云学术

Distribution and progression of cerebral amyloid angiopathy in early-onset V30M (p.V50M) hereditary ATTR amyloidosis

Published:2022-09-30 Issue:1 Volume:30 Page:109-118
ISSN:1350-6129
Container-title:Amyloid
language:en
Short-container-title:Amyloid

Author:

Takahashi Yusuke¹,Oguchi Kazuhiro²,Mochizuki Yusuke¹,Takasone Ken¹,Ezawa Naoki¹,Matsushima Akira³,Katoh Nagaaki¹,Yazaki Masahide⁴⁵,Sekijima Yoshiki¹²⁵^ORCID

Affiliation:

1. Department of Medicine (Neurology and Rheumatology), Shinshu University School of Medicine, Matsumoto, Japan

2. Jisenkai Brain Imaging Research Center, Matsumoto, Japan

3. Department of Neurology, JA Nagano Koseiren Kakeyu Misayama Rehabilitation Center Kakeyu Hospital, Matsumoto, Japan

4. Department of Biomedical Laboratory Sciences, Shinshu University School of Health Sciences, Matsumoto, Japan

5. Institute for Biomedical Sciences, Shinshu University, Matsumoto, Japan

Funder

Grant-in-aid for Scientific Research

Grant from the Amyloidosis Research Committee

Publisher

Informa UK Limited

Subject

Internal Medicine

Link

https://www.tandfonline.com/doi/pdf/10.1080/13506129.2022.2128331

Reference48 articles.

1. Hereditary transthyretin amyloidosis: a model of medical progress for a fatal disease

2. Transthyretin (ATTR) amyloidosis: clinical spectrum, molecular pathogenesis and disease-modifying treatments

3. The Biological and Chemical Basis for Tissue-Selective Amyloid Disease

4. Sequence-dependent denaturation energetics: A major determinant in amyloid disease diversity

5. Clinical improvement and amyloid regression after liver transplantation in hereditary transthyretin amyloidosis

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