Predictors of cognitive dysfunction in hereditary transthyretin amyloidosis with liver transplant-Reference-Cited by-同舟云学术

Predictors of cognitive dysfunction in hereditary transthyretin amyloidosis with liver transplant

Published:2022-10-17 Issue:1 Volume:30 Page:119-126
ISSN:1350-6129
Container-title:Amyloid
language:en
Short-container-title:Amyloid

Author:

Cavaco Sara¹²³,Martins da Silva Ana²³⁴,Fernandes Joana⁴,Sousa Ana Paula⁴,Alves Cristina⁴,Cardoso Márcio⁴,Teixeira-Pinto Armando⁵^ORCID,Coelho Teresa⁴

Affiliation:

1. Neuropsychology Unit, Centro Hospitalar Universitário do Porto, Porto, Portugal

2. UMIB – Unit for Multidisciplinary Research in Biomedicine, ICBAS – School of Medicine and Biomedical Sciences, University of Porto, Porto, Portugal

3. ITR – Laboratory for Integrative and Translational Research in Population Health, Porto, Portugal

4. Corino de Andrade Unit, Centro Hospitalar Universitário do Porto, Porto, Portugal

5. Sydney School of Public Health, The University of Sydney, Sydney, Australia

Publisher

Informa UK Limited

Subject

Internal Medicine

Link

https://www.tandfonline.com/doi/pdf/10.1080/13506129.2022.2131384

Reference19 articles.

1. Transthyretin (ATTR) amyloidosis: clinical spectrum, molecular pathogenesis and disease-modifying treatments

Diagnosis and Screening of Patients with Hereditary Transthyretin Amyloidosis (hATTR): Current Strategies and Guidelines

3. A PECULIAR FORM OF PERIPHERAL NEUROPATHY

4. Expert consensus recommendations to improve diagnosis of ATTR amyloidosis with polyneuropathy

5. Overcoming artefact: anticipation in 284 Portuguese kindreds with familial amyloid polyneuropathy (FAP) ATTRV30M

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Hereditary transthyretin amyloid neuropathies: advances in pathophysiology, biomarkers, and treatment;The Lancet Neurology;2023-11