Overexpression of PLK1 relieved the myocardial ischemia-reperfusion injury of rats through inducing the mitophagy and regulating the p-AMPK/FUNDC1 axis-Reference-Cited by-同舟云学术

Overexpression of PLK1 relieved the myocardial ischemia-reperfusion injury of rats through inducing the mitophagy and regulating the p-AMPK/FUNDC1 axis

Published:2021-01-01 Issue:1 Volume:12 Page:2676-2687
ISSN:2165-5979
Container-title:Bioengineered
language:en
Short-container-title:Bioengineered

Author:

Mao Shan¹,Tian Shuning²,Luo Xianghong³,Zhou Ming¹,Cao Zheng¹,Li Ji⁴

Affiliation:

1. Department of Cardiology, Taihe Hospital, Hubei University of Medicine, Shiyan City, Hubei Province, China

2. Department of Anesthesiology, Jiangmen Central Hospital Affiliated Jiangmen Hospital of Sun Yat-sen University, Jiangmen City, Guangdong Province, China

3. Department of Anesthesiology, Taihe Hospital, Hubei University of Medicine, Shiyan City, Hubei Province, China

4. Department of Cardiovascular Internal Medicine, The Second Affiliated Hospital of Guizhou Medical University, Kaili City, Guizhou Province, China

Funder

funding to report

Publisher

Informa UK Limited

Subject

Applied Microbiology and Biotechnology,General Medicine,Bioengineering,Biotechnology

Link

https://www.tandfonline.com/doi/pdf/10.1080/21655979.2021.1938500

Reference45 articles.

1. LncRNA CAIF inhibits autophagy and attenuates myocardial infarction by blocking p53-mediated myocardin transcription

2. APF lncRNA regulates autophagy and myocardial infarction by targeting miR-188-3p

3. Dihydropyridines’ metabolites-induced early apoptosis after myocardial infarction in rats; new outlook on preclinical study with M-2 and M-3

4. Cardiomyocyte death in ischemia-reperfusion is mediated by opening of the mitochondrial permeability transition pore: Role of oxidant stress during ischemia

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