Autocrine-paracrine inhibition of growth hormone and prolactin production by GH3 cell-conditioned medium-Reference-Cited by-同舟云学术

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Autocrine-paracrine inhibition of growth hormone and prolactin production by GH3 cell-conditioned medium

Published:1989-06 Issue:6 Volume:25 Page:528-534
ISSN:0883-8364
Container-title:In Vitro Cellular & Developmental Biology
language:en
Short-container-title:In Vitro Cell Dev Biol

Author:

Lapp C. A.,Tyler J. M.,Lee Y. S.,Stachura M. E.

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Clinical Biochemistry,Developmental Biology,Plant Science,Biotechnology

Link

http://link.springer.com/content/pdf/10.1007/BF02623565.pdf

Reference28 articles.

1. Abe, H.; Engler, D.; Molitch, M., et al. Vasoactive intestinal peptide is a physiological mediator of PRL release in the rat. Endocrinology 116:1383–1390; 1985.

2. Bentley, A. M.; Wallis, M.In-vitro evidence for the autoregulation of prolactin secretion at the level of the pituitary gland in the rat. J. Endocrinol. 115:13–18; 1987.

3. Boockfor, F. R.; Schwarz, L. K. Cultures of GH3 cells contain both single and dual hormone secretors. Endocrinology 122:762–764; 1988.

4. Boockfor, F. R.; Hoeffler, J. P.; Frawley, L. S. Cultures of GH3 cells are functionally heterogeneous: thyrotropin-releasing hormone, estradiol and cortisol cause reciprocal shifts in the proportions of growth hormone and prolactin secretors. Endocrinology 117:418–420; 1985.

5. Ceda, G. P.; Hoffman, A. R.; Silverberg, G. D., et al. Regulation of growth hormone release from cultured human pituitary adenomas by somatomedins and insulin. J. Clin. Endocrinol. Metab. 60:1204–1209; 1985.

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