The Role of JAKs and STAT3 in Regulation of Regenerative-Competent Cells of the Nervous Tissue in β-Amyloid-Induced Neurodegeneration
Author:
Publisher
Springer Science and Business Media LLC
Subject
General Biochemistry, Genetics and Molecular Biology,General Medicine
Link
https://link.springer.com/content/pdf/10.1007/s10517-022-05560-w.pdf
Reference15 articles.
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2. Bernabeu-Zornoza A, Coronel R, Palmer C, Monteagudo M, Zambrano A, Liste I. Physiological and pathological effects of amyloid-β species in neural stem cell biology. Neural Regen. Res. 2019;14(12):2035-2042. https://doi.org/10.4103/1673-5374.262571
3. Kaeser G, Chun J. Brain cell somatic gene recombination and its phylogenetic foundations. J. Biol. Chem. 2020;295(36):12 786-12 795. https://doi.org/10.1074/jbc.REV120.009192
4. Lu J, Li Y, Mollinari C, Garaci E, Merlo D, Pei G. Amyloid-β oligomers-induced mitochondrial DNA repair impairment contributes to altered human neural stem cell differentiation. Curr. Alzheimer Res. 2019;16(10):934-949. https://doi.org/10.2174/1567205016666191023104036
5. Ohgomori T, Jinno S. Signal transducer and activator of transcription 3 activation in hippocampal neural stem cells and cognitive deficits in mice following short-term cuprizone exposure. Neuroscience. 2021;472:90-102. https://doi.org/10.1016/j.neuroscience.2021.07.031
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