Simulations of postvagal tachycardia at the single cell pacemaker level: A new hypothesis
Author:
Publisher
Springer Science and Business Media LLC
Subject
Biomedical Engineering
Link
http://link.springer.com/content/pdf/10.1007/BF02684161.pdf
Reference40 articles.
1. Alter W. A. III, G. K. Weiss, and D. V. Priola. Vagally induced tachycardia in atropinized dogs. effect of β-adrenergic blockade.Eur. J. Pharmacol. 24:329–333, 1973.
2. Bouman, L. N., E. D. Gerlings, P. A. Biersteker, and F. I. Bonke. Pacemaker shift in the sinoatrial node during vagal stimulation.Pflügers Arch. 302:255–267, 1968.
3. Boyett, M. R., and A. Roberts. The fade of the response to acetylcholine at the rabbit isolated sino-atrial node.J. Physiol. (Lond.) 393:171–194, 1987.
4. Breitwieser, G. E., and G. Szabo, Mechanism of muscarinic receptor-induced K+ channel activation as revealed by hydrolysis-resistant GTP analogues.J. Gen. Physiol. 91:469–493, 1988.
5. Brown, G. L., and J. C. Eccles. The action of a single vagal volley on the rhythm of the heart beat.J. Physiol. (Lond.) 82:211–241, 1934.
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