Global transcriptomic response of the AI-3 isomers 3,5-DPO and 3,6-DPO in Salmonella Typhimurium
Author:
Funder
Novo Nordisk Fonden
Publisher
Springer Science and Business Media LLC
Subject
Genetics,Molecular Biology,General Medicine,Biochemistry,Microbiology
Link
https://link.springer.com/content/pdf/10.1007/s00203-023-03450-x.pdf
Reference49 articles.
1. Abdulla SZ, Kim K, Azam MS et al (2022) Small RNAs activate salmonella pathogenicity island 1 by modulating mRNA stability through the hilD mRNA 3’ untranslated region. J Bacteriol. https://doi.org/10.1128/jb.00333-22
2. Bassler BL (2002) Small talk. Cell-to-cell communication in bacteria. Cell 109:421–424. https://doi.org/10.1016/s0092-8674(02)00749-3
3. Bearson BL, Bearson SMD (2008) The role of the QseC quorum-sensing sensor kinase in colonization and norepinephrine-enhanced motility of Salmonella enterica serovar Typhimurium. Microb Pathog 44:271–278. https://doi.org/10.1016/j.micpath.2007.10.001
4. Cheverton AM, Gollan B, Przydacz M et al (2016) A Salmonella toxin promotes persister formation through acetylation of tRNA. Mol Cell 63:86–96. https://doi.org/10.1016/j.molcel.2016.05.002
5. Clarke MB, Sperandio V (2005) Transcriptional autoregulation by quorum sensing Escherichia coli regulators B and C (QseBC) in enterohaemorrhagic E. coli (EHEC). Mol Microbiol 58:441–455. https://doi.org/10.1111/j.1365-2958.2005.04819.x
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