Dual roles of UFMylation on stalling fork stability
Author:
Funder
National Natural Science Foundation of China
Key Technologies Research and Development Program of Anhui Province
Publisher
Springer Science and Business Media LLC
Link
https://link.springer.com/content/pdf/10.1007/s42764-024-00129-5.pdf
Reference27 articles.
1. Amunugama, R., Willcox, S., Wu, R. A., Abdullah, U. B., El-Sagheer, A. H., Brown, T., McHugh, P. J., Griffith, J. D., & Walter, J. C. (2018). Replication fork reversal during DNA interstrand Crosslink Repair requires CMG unloading. Cell Rep, 23, 3419–3428. https://doi.org/10.1016/j.celrep.2018.05.061.
2. Berti, M., Cortez, D., & Lopes, M. (2020). The plasticity of DNA replication forks in response to clinically relevant genotoxic stress. Nature Reviews Molecular Cell Biology, 21, 633–651. https://doi.org/10.1038/s41580-020-0257-5.
3. Cong, K., & Cantor, S. B. (2022). Exploiting replication gaps for cancer therapy. Molecular Cell, 82, 2363–2369. https://doi.org/10.1016/j.molcel.2022.04.023.
4. Cortez, D. (2019). Replication-coupled DNA repair. Molecular Cell, 74, 866–876. https://doi.org/10.1016/j.molcel.2019.04.027.
5. Gong, Y., Wang, Z., Zong, W., Shi, R., Sun, W., Wang, S., Peng, B., Takeda, S., Wang, Z. Q., & Xu, X. (2024). PARP1 UFMylation ensures the stability of stalled replication forks. Proc Natl Acad Sci U S A, 121, e2322520121.
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