Human Amnion Membrane Proteins Prevent Doxorubicin-Induced Oxidative Stress Injury and Apoptosis in Rat H9c2 Cardiomyocytes
Author:
Funder
Tabriz University of Medical Sciences
Publisher
Springer Science and Business Media LLC
Subject
Cardiology and Cardiovascular Medicine,Toxicology,Molecular Biology
Link
http://link.springer.com/content/pdf/10.1007/s12012-020-09564-8.pdf
Reference42 articles.
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2. Bose, C., Awasthi, S., Sharma, R., Benes, H., Hauer-Jensen, M., Boerma, M., et al. (2018). Sulforaphane potentiates anticancer effects of doxorubicin and attenuates its cardiotoxicity in a breast cancer model. PLoS ONE,13, e0193918.
3. Chatterjee, K., Zhang, J., Honbo, N., & Karliner, J. S. (2010). Doxorubicin cardiomyopathy. Cardiology,115, 155–162.
4. Zhu, H., Gao, Y., Zhu, S., Cui, Q., & Du, J. (2017). Klotho improves cardiac function by suppressing reactive oxygen species (ROS) mediated apoptosis by modulating Mapks/Nrf2 signaling in doxorubicin-induced cardiotoxicity. Medical Science Monitor,23, 5283–5293.
5. Zhang, Q. L., Yang, J. J., & Zhang, H. S. (2019). Carvedilol (CAR) combined with carnosic acid (CAA) attenuates doxorubicin-induced cardiotoxicity by suppressing excessive oxidative stress, inflammation, apoptosis and autophagy. Biomedicine & Pharmacotherapy,109, 71–83.
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