Stress, Vascular Smooth Muscle Cell Phenotype and Atherosclerosis: Novel Insight into Smooth Muscle Cell Phenotypic Transition in Atherosclerosis
Author:
Funder
National Nature Science Foundation of China
Shandong Provincial Natural Science Foundation
Publisher
Springer Science and Business Media LLC
Link
https://link.springer.com/content/pdf/10.1007/s11883-024-01220-8.pdf
Reference150 articles.
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2. Yu Q, Li W, Jin R, Yu S, Xie D, Zheng X, et al. PI3Kγ (Phosphoinositide 3-Kinase γ) regulates vascular smooth muscle cell phenotypic modulation and neointimal formation through CREB (Cyclic AMP-Response Element Binding Protein)/YAP (Yes-Associated Protein) signaling. Arterioscler Thromb Vasc Biol. 2019;39:e91-105.
3. Pan H, Ho SE, Xue C, Cui J, Johanson QS, Sachs N, et al. Atherosclerosis is a smooth muscle cell-driven tumor-like disease. Circulation. 2024. https://doi.org/10.1161/CIRCULATIONAHA.123.067587
4. Owens GK, Kumar MS, Wamhoff BR. Molecular regulation of vascular smooth muscle cell differentiation in development and disease. Physiol Rev. 2004;84:767–801.
5. Wang Z-Y, Cheng J, Liu B, Xie F, Li C-L, Qiao W, et al. Protein deglycase DJ-1 deficiency induces phenotypic transitioning in vascular smooth muscle cells and exacerbates atherosclerotic plaque instability. J Cell Mol Med. 2021;25:2816–27.
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