β-Amyloid 25-35 Peptide Reduces the Expression of Glutamine Transporter SAT1 in Cultured Cortical Neurons-Reference-Cited by-同舟云学术

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β-Amyloid 25-35 Peptide Reduces the Expression of Glutamine Transporter SAT1 in Cultured Cortical Neurons

Published:2007-11-01 Issue:2 Volume:33 Page:248-256
ISSN:0364-3190
Container-title:Neurochemical Research
language:en
Short-container-title:Neurochem Res

Author:

Buntup Doungjai,Skare Øivind,Solbu Tom Tallak,Chaudhry Farrukh A.,Storm-Mathisen Jon,Thangnipon Wipawan

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,General Medicine,Biochemistry

Link

https://link.springer.com/content/pdf/10.1007/s11064-007-9527-2.pdf

Reference28 articles.

1. Mattson MP (2004) Pathways towards and away from Alzheimer’s disease. Nature 430:631–639

2. Goedert M, Spillantini MG (2006) A century of Alzheimer’s disease. Science 314:777–781

3. Kasa P, Papp H, Kasa Jr P et al (2004) Effects of amyloid-beta on cholinergic and acetylcholinesterase-positive cells in cultured basal forebrain neurons of embryonic rat brain. Brain Res 998:73–82

4. Mattson MP, Cheng B, Davis D et al (1992) β-Amyloid peptides destabilize calcium homeostasis and render human cortical neurons vulnerable to excitotoxicity. J Neurosci 12:376–389

5. Varadarajan S, Kanski J, Aksenova M et al (2001). Different mechanisms of oxidative stress and neurotoxicity for Alzheimer’s Aβ(1–42) and Aβ(25-35). J Am Chem Soc 123:5625–5631

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