TRPM2 Cation Channels, Oxidative Stress and Neurological Diseases: Where Are We Now?
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cellular and Molecular Neuroscience,General Medicine,Biochemistry
Link
http://link.springer.com/content/pdf/10.1007/s11064-010-0347-4.pdf
Reference67 articles.
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3. Kühn FJ, Heiner I, Luckhoff A (2005) TRPM2: a calcium influx pathway regulated by oxidative stress and the novel second messenger ADP-ribose. Pflugers Arch 451:212–219
4. Hara Y, Wakamori M, Ishii M, Maeno E, Nishida M, Yoshida T, Yamada H, Shimizu S, Mori E, Kudoh J, Shimizu N, Kurose H, Okada Y, Imoto K, Mori Y (2002) LTRPC2 Ca2+-permeable channel activated by changes in redox status confers susceptibility to cell death. Mol Cell 9:163–173
5. Heiner I, Eisfeld J, Warnstedt M, Radukina N, Jungling E, Luckhoff A (2006) Endogenous ADP-ribose enables calcium-regulated cation currents through TRPM2 channels in neutrophil granulocytes. Biochem J 398:225–232
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