Inhibition of cardiomyocyte automaticity by electrotonic application of inward rectifier current from Kir2.1 expressing cells-Reference-Cited by-同舟云学术

Inhibition of cardiomyocyte automaticity by electrotonic application of inward rectifier current from Kir2.1 expressing cells

Published:2006-04-20 Issue:7 Volume:44 Page:537-542
ISSN:0140-0118
Container-title:Medical and Biological Engineering and Computing
language:en
Short-container-title:Med Bio Eng Comput

Author:

de Boer Teun P.,van Veen Toon A. B.,Houtman Marien J. C.,Jansen John A.,van Amersfoorth Shirley C. M.,Doevendans Pieter A.,Vos Marc A.,van der Heyden Marcel A. G.

Publisher

Springer Science and Business Media LLC

Subject

Computer Science Applications,Biomedical Engineering

Link

http://link.springer.com/content/pdf/10.1007/s11517-006-0059-8.pdf

Reference24 articles.

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2. Dhamoon AS, Jalife J (2005) The inward rectifier current (I K1) controls cardiac excitability and is involved in arrhythmogenesis. Heart Rhythm 2:316–324. DOI 10.1016/j.hrthm.2004.11.012

3. Harvey DM, Caskey CT (1998) Inducible control of gene expression: prospects for gene therapy. Curr Opin Chem Biol 2:512–518. DOI 10.1016/S1367-5931(98)80128-2

4. Koumi S, Backer CL, Arentzen CE, Sato R (1995) β-Adrenergic modulation of the inwardly rectifying potassium channel in isolated human ventricular myocytes. Alteration in channel response to β-adrenergic stimulation in failing human hearts. J Clin Invest 96:2870–2881

5. Lange PS, Er F, Gassanov N, Hoppe UC (2003) Andersen mutations of KCNJ2 suppress the native inward rectifier current I K1 in a dominant-negative fashion. Cardiovasc Res 59:321–327. DOI 10.1016/S0008-6363(03)00434-6

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