Author:
Hermenegildo Carlos,Marcaida Goizane,Montoliu Carmina,Grisolía Santiago,Miñana María-Dolores,Felipo Vicente
Publisher
Springer Science and Business Media LLC
Subject
Cellular and Molecular Neuroscience,General Medicine,Biochemistry
Reference76 articles.
1. Moroni, F., Lombardi, G., Moneti, G., and Cortesini, C. 1983. The release and neosynthesis of glutamic acid are increased in experimental model of hepatic encephalopathy. J. Neurochem. 40: 850–854.
2. Moroni, F., Lombardi, G., Moneti, G., Pellegrini, D., and Cortesini, C. 1994. A possible role for excitatory neurotoxic amino acids in the pathogenesis of hepatic encephalopathy. Pages 41–51,in J. E. Fischer and F. Rossi-Fanelli, (eds.), Hepatic encephalopathy in chronic liver failure, ed. by L. Capocaccia, Plenum Publishing Co. New York.
3. Moroni, F., Lombardi, G., Carlá, V., Lal, S., Etienne, P., and Nair, N. P. V. 1986. Increase in the content of quinolinic acid in cerebrospinal fluid and frontal cortex of patients with hepatic failure. J. Neurochem. 47:1667–1671.
4. Moroni, F., Lombardi, G., Carlà, V., Pellegrini, D., Carassale, G. L., and Cortesini, C. 1986. Content of quinolinic acid and of other tryptophan metabolites increases in brain regions of rats used as experimental models of hepatic encephalopathy. J. Neurochem. 46:869–874.
5. Marcaida, G., Felipo, V., Hermenegildo, C., Miñana, M. D., and Grisolía, S. 1992. Acute ammonia toxicity is mediated by the NMDA type of glutamate receptors. FEBS Lett. 296:67–68.
Cited by
134 articles.
订阅此论文施引文献
订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献