Smad linker region phosphorylation is a signalling pathway in its own right and not only a modulator of canonical TGF-β signalling
Author:
Funder
National Health and Medical Research Council
National Heart Foundation of Australia
Publisher
Springer Science and Business Media LLC
Subject
Cell Biology,Cellular and Molecular Neuroscience,Pharmacology,Molecular Biology,Molecular Medicine
Link
http://link.springer.com/content/pdf/10.1007/s00018-019-03266-3.pdf
Reference58 articles.
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2. Frick CL et al (2017) Sensing relative signal in the Tgf-beta/Smad pathway. Proc Natl Acad Sci USA 114(14):E2975–E2982
3. Massague J, Seoane J, Wotton D (2005) Smad transcription factors. Genes Dev 19(23):2783–2810
4. Dennler S, Huet S, Gauthier JM (1999) A short amino-acid sequence in MH1 domain is responsible for functional differences between Smad2 and Smad3. Oncogene 18(8):1643–1648
5. Liu L et al (2016) Smad2 and Smad3 have differential sensitivity in relaying TGFbeta signaling and inversely regulate early lineage specification. Sci Rep 6:21602
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