Further Studies on the Hypothesis of PARP-1 Inhibition as a Strategy for Lessening the Long-Term Effects Produced by Perinatal Asphyxia: Effects of Nicotinamide and Theophylline on PARP-1 Activity in Brain and Peripheral Tissue-Reference-Cited by-同舟云学术

Further Studies on the Hypothesis of PARP-1 Inhibition as a Strategy for Lessening the Long-Term Effects Produced by Perinatal Asphyxia: Effects of Nicotinamide and Theophylline on PARP-1 Activity in Brain and Peripheral Tissue

Published:2012-02-04 Issue:1 Volume:22 Page:79-90
ISSN:1029-8428
Container-title:Neurotoxicity Research
language:en
Short-container-title:Neurotox Res

Author:

Allende-Castro C.,Espina-Marchant P.,Bustamante D.,Rojas-Mancilla E.,Neira T.,Gutierrez-Hernandez M. A.,Esmar D.,Valdes J. L.,Morales P.,Gebicke-Haerter P. J.,Herrera-Marschitz M.

Publisher

Springer Science and Business Media LLC

Subject

Toxicology,General Neuroscience

Link

http://link.springer.com/content/pdf/10.1007/s12640-012-9310-2.pdf

Reference45 articles.

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3. Bustamante D, Goiny M, Åström G, Gross J, Andersson K, Herrera-Marschitz M (2003) Nicotinamide prevents the long-term effects of perinatal asphyxia on basal ganglia monoamine systems in the rat. Exp Brain Res 148:227–232

4. Bustamante D, Morales P, Torres-Pereyra J, Goiny M, Herrera-Marschitz M (2007) Nicotinamide prevents the effect of perinatal asphyxia on dopamine release evaluated with in vivo microdialysis 3 months after birth. Exp Brain Res 177:358–369

5. Chen Y, Engidawork E, Loidl F, Dell’Anna E, Goiny M, Lubec G, Andersson K, Herrera-Marschitz M (1997) Short- and long-term effects of perinatal asphyxia on monoamine, amino acid and glycolysis product levels measured in the basal ganglia of the rat. Dev Brain Res 104:19–30

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