NO Orchestrates the Loss of Synaptic Boutons from Adult “Sick” Motoneurons: Modeling a Molecular Mechanism-Reference-Cited by-同舟云学术

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NO Orchestrates the Loss of Synaptic Boutons from Adult “Sick” Motoneurons: Modeling a Molecular Mechanism

Published:2010-12-29 Issue:1 Volume:43 Page:41-66
ISSN:0893-7648
Container-title:Molecular Neurobiology
language:en
Short-container-title:Mol Neurobiol

Author:

Moreno-López Bernardo,Sunico Carmen R.,González-Forero David

Publisher

Springer Science and Business Media LLC

Subject

Neuroscience (miscellaneous),Cellular and Molecular Neuroscience,Neurology

Link

http://link.springer.com/content/pdf/10.1007/s12035-010-8159-8.pdf

Reference244 articles.

1. Palop JJ, Chin J, Mucke L (2006) A network dysfunction perspective on neurodegenerative diseases. Nature 443:768–773

2. Small DH (2008) Network dysfunction in Alzheimer's disease: does synaptic scaling drive disease progression? Trends Mol Med 14:103–108

3. Scheff SW, Price DA (2006) Alzheimer's disease-related alterations in synaptic density: neocortex and hippocampus. J Alzheimers Dis 9:101–115

4. Emre M (2003) Dementia associated with Parkinson's disease. Lancet Neurol 2:229–237

5. Day M, Wang Z, Ding J et al (2006) Selective elimination of glutamatergic synapses on striatopallidal neurons in Parkinson disease models. Nat Neurosci 9:251–259

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3. Long-Term Suppression of c-Jun and nNOS Preserves Ultrastructural Features of Lower Motor Neurons and Forelimb Function after Brachial Plexus Roots Avulsion;Cells;2021-06-28

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