A new mutation inEscherichia coli K12,isfA, which is responsible for inhibition of SOS functions
Author:
Publisher
Springer Science and Business Media LLC
Subject
Genetics,Molecular Biology
Link
http://link.springer.com/content/pdf/10.1007/BF02456619.pdf
Reference46 articles.
1. Bagdasarian M, Bailone A, Bagdasarian MM, Manning, PA, Lurz R, Timmis KN, Devoret R (1986) An inhibitor of SOS induction specified by a plasmid locus inEscherichia coli. Proc Natl Acad Sci USA 83:5723–5726
2. Bagdasarian M, Bailone A, Angulo JF, Stolz P, Bagdasarian M, Devoret R (1992) PsiB, an anti-SOS protein is transiently expressed by the F sex factor during its transmission to anEscherichia coli K-12 recipient. Mol Microbiol 6(7):885–893
3. Bates H, Bridges BA (1991) Mutagenic DNA repair inEscherichia coli. XIX On the roles of RecA protein in ultraviolet light mutagenesis. Biochemie 73:485–489
4. Bates H, Randall SK, Rayssiguier Ch, Bridges BA, Goodman MF, Radman M (1989) Spontaneous and UV-induced mutations inEscherichia coli K-12 strains with altered or absent DNA polymerase I. J Bacteriol 171:2480–2484
5. Bębenek K, Janion C (1985) Ability of base analogs to induce the SOS response: effect of adam mutation and mismatch repair system. Mol Gen Genet 201:519–524
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3. A model for the abrogation of the SOS response by an SOS protein: a negatively charged helix in DinI mimics DNA in its interaction with RecA;Genes & Development;2001-02-15
4. The isfA mutation specifically inhibits the SOS-dependent mutagenic pathway and does not selectively affect any particular base substitution;Mutagenesis;1999-05-01
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