Complement activation by phospholipids: the interplay of factor H and C1q

Author:

Tan Lee Aun,Yu Bingbin,Sim Francis C. J.,Kishore Uday,Sim Robert B.

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Drug Discovery,Biochemistry,Biotechnology

Reference64 articles.

1. Agnello, V., Carr, R.I., Koffler, D., and Kunkel, H. (1969). Gel diffusion reactions of C1 with aggregated globulin, DNA and various anionic substances. Fed Proc 28, 696.

2. Bradley, A.J., Brooks, D.E., Norris-Jones, R., and Devine, D.V. (1999). C1q binding to liposomes is surface charge dependent and is inhibited by peptides consisting of residues 14–26 of the human C1qA chain in a sequence independent manner. Biochim Biophys Acta 1418, 19–30.

3. Carreno, M.P., Labarre, D., Maillet, F., Jozefowicz, M., and Kazatchkine, M.D. (1989). Regulation of the human alternative complement pathway: formation of a ternary complex between factor H, surface-bound C3b and chemical groups on nonactivating surfaces. Eur J Immunol 19, 2145–2150.

4. Charlesworth, J.A., Scott, D.M., Pussell, B.A., and Peters, D.K. (1979). Metabolism of human beta 1H: studies in man and experimental animals. Clin Exp Immunol 38, 397–404.

5. Chonn, A., Cullis, P.R., and Devine, D.V. (1991). The role of surface charge in the activation of the classical and alternative pathways of complement by liposomes. J Immunol 146, 4234–4241.

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