Dopaminergic midbrain neurons are the prime target for mitochondrial DNA deletions-Reference-Cited by-同舟云学术

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Dopaminergic midbrain neurons are the prime target for mitochondrial DNA deletions

Published:2008-07-11 Issue:8 Volume:255 Page:1231-1235
ISSN:0340-5354
Container-title:Journal of Neurology
language:en
Short-container-title:J Neurol

Author:

Bender A.,Schwarzkopf R.-M.,McMillan A.,Krishnan K. J.,Rieder G.,Neumann M.,Elstner M.,Turnbull D. M.,Klopstock T.

Publisher

Springer Science and Business Media LLC

Subject

Neurology (clinical),Neurology

Link

http://link.springer.com/content/pdf/10.1007/s00415-008-0892-9.pdf

Reference36 articles.

1. Anandatheerthavarada HK, Biswas G, Robin MA, Avadhani NG (2003) Mitochondrial targeting and a novel transmembrane arrest of Alzheimer’s amyloid precursor protein impairs mitochondrial function in neuronal cells. J Cell Biol 161:41–54

2. Atamna H, Frey WH 2nd (2007) Mechanisms of mitochondrial dysfunction and energy deficiency in Alzheimer’s disease. Mitochondrion 7:297–310

3. Bender A, Krishnan KJ, Morris CM, Taylor GA, Reeve AK, Perry RH, Jaros E, Hersheson JS, Betts J, Klopstock T, Taylor RW, Turnbull DM (2006) High levels of mitochondrial DNA deletions in substantia nigra neurons in aging and Parkinson disease. Nat Genet 38:515–517

4. Betarbet R, Sherer TB, MacKenzie G, Garcia-Osuna M, Panov AV, Greenamyre JT (2000) Chronic systemic pesticide exposure reproduces features of Parkinson‘s disease. Nat Neurosci 3:1301–1306

5. Betts J, Lightowlers RN, Turnbull DM (2004) Neuropathological aspects of mitochondrial DNA disease. Neurochem Res 29:505–511

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