Blocking angiotensin 2 receptor attenuates diabetic nephropathy via mitigating ANGPTL2/TL4/NF-κB expression
Author:
Publisher
Springer Science and Business Media LLC
Subject
Genetics,Molecular Biology,General Medicine
Link
https://link.springer.com/content/pdf/10.1007/s11033-021-06647-9.pdf
Reference50 articles.
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2. Lin YC, Chang YH, Yang SY, Wu KD, Chu TS (2018) Update of pathophysiology and management of diabetic kidney disease. J Formos Med Assoc 117:662–675. https://doi.org/10.1016/j.jfma.2018.02.007
3. Cao Z, Cooper ME (2011) Pathogenesis of diabetic nephropathy. J Diabetes Investig 2:243–247. https://doi.org/10.1111/j.2040-1124.2011.00131.x
4. Patinha D, Fasching A, Pinho D, Albino-Teixeira A, Morato M, Palm F (2013) Angiotensin II contributes to glomerular hyperfiltration in diabetic rats independently of adenosine type I receptors. Am J Physiol Renal Physiol 304:F614–F622. https://doi.org/10.1152/ajprenal.00285.2012
5. Tuttle KR (2017) Back to the future: glomerular hyperfiltration and the diabetic kidney. Diabetes 66:14–16. https://doi.org/10.2337/dbi16-0056
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