A revised method of troponin exchange in permeabilised cardiac trabeculae using vanadate: functional consequences of a HCM-causing mutation in troponin I-Reference-Cited by-同舟云学术

A revised method of troponin exchange in permeabilised cardiac trabeculae using vanadate: functional consequences of a HCM-causing mutation in troponin I

Published:2006-10-19 Issue:8 Volume:27 Page:585-590
ISSN:0142-4319
Container-title:Journal of Muscle Research and Cell Motility
language:en
Short-container-title:J Muscle Res Cell Motil

Author:

Preston Laura C.,Watkins Hugh,Redwood Charles S.

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Biochemistry,Physiology

Link

http://link.springer.com/content/pdf/10.1007/s10974-006-9079-0.pdf

Reference21 articles.

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3. Brenner B, Kraft T, Yu LC, Chalovich JM (1999) Thin filament activation probed by fluorescence of N-((2-(iodoacetoxy)ethyl)-N-methyl)amino-7-nitrobenz-2-oxa-1,3-diazole-labeled troponin I incorporated into skinned fibers of rabbit psoas muscle. Biophys J 77:2677–2691

4. Burton D, Abdulrazzak H, Knott A, Elliott K, Redwood C, Watkins H, Marston S, Ashley C (2002) Two mutations in troponin I that cause hypertrophic cardiomyopathy have contrasting effects on cardiac muscle contractility. Biochem J 362:443–451

5. Deng Y, Schmidtmann A, Kruse S, Filatov V, Heilmeyer LM Jr, Jaquet K, Thieleczek R (2003) Phosphorylation of human cardiac troponin I G203S and K206Q linked to familial hypertrophic cardiomyopathy affects actomyosin interaction in different ways. J Mol Cell Cardiol 35:1365–1374

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