SHP2 Targets ITK Downstream of PD-1 to Inhibit T Cell Function
Author:
Funder
National Institutes of Health
Cancer Research Institute
Publisher
Springer Science and Business Media LLC
Subject
Immunology,Immunology and Allergy
Link
https://link.springer.com/content/pdf/10.1007/s10753-021-01437-8.pdf
Reference29 articles.
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3. Hui, E., J. Cheung, J. Zhu, X. Su, M.J. Taylor, H.A. Wallweber, D.K. Sasmal, J. Huang, J.M. Kim, I. Mellman, and R.D. Vale. 2017. T cell costimulatory receptor CD28 is a primary target for PD-1-mediated inhibition. Science. 355 (6332): 1428–1433. https://doi.org/10.1126/science.aaf1292.
4. Chemnitz, J.M., R.V. Parry, K.E. Nichols, C.H. June, and J.L. Riley. 2004. SHP-1 and SHP-2 associate with immunoreceptor tyrosine-based switch motif of programmed death 1 upon primary human T cell stimulation, but only receptor ligation prevents T cell activation. Journal of Immunology 173 (2): 945–954. https://doi.org/10.4049/jimmunol.173.2.945.
5. Celis-Gutierrez, J., P. Blattmann, Y. Zhai, N. Jarmuzynski, K. Ruminski, C. Grégoire, Y. Ounoughene, F. Fiore, R. Aebersold, R. Roncagalli, M. Gstaiger, and B. Malissen. 2019. Quantitative interactomics in primary T cells provides a rationale for concomitant PD-1 and BTLA coinhibitor blockade in cancer immunotherapy. Cell Reports 27 (11): 3315–3330.e7. https://doi.org/10.1016/j.celrep.2019.05.041.
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