SOCS3 Silencing Promotes Activation of Vocal Fold Fibroblasts via JAK2/STAT3 Signaling Pathway-Reference-Cited by-同舟云学术

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SOCS3 Silencing Promotes Activation of Vocal Fold Fibroblasts via JAK2/STAT3 Signaling Pathway

Published:2023-05-08 Issue:4 Volume:46 Page:1318-1331
ISSN:0360-3997
Container-title:Inflammation
language:en
Short-container-title:Inflammation

Author:

Li Xueyan,Hu Rong,Wang Haizhou,Xu Wen

Funder

National Natural Science Foundation of China

Natural Science Foundation of Beijing Municipality

Publisher

Springer Science and Business Media LLC

Subject

Immunology,Immunology and Allergy

Link

https://link.springer.com/content/pdf/10.1007/s10753-023-01810-9.pdf

Reference31 articles.

1. Meng, X.M., D.J. Nikolic-Paterson, and H.Y. Lan. 2016. TGF-beta: The master regulator of fibrosis. Nature Reviews. Nephrology 12 (6): 325–338.

2. Kuzet, S., and C. Gaggioli. 2016. Fibroblast activation in cancer: When seed fertilizes soil. Cell and tissue research 365 (3): 607–619.

3. Zeisberg, E.M., and M. Zeisberg. 2013. The role of promoter hypermethylation in fibroblast activation and fibrogenesis. The Journal of Pathology 229 (2): 264–273.

4. Frangogiannis, N. 2021. Cardiac fibrosis. Cardiovascular research 117 (6): 1450–1488.

5. Hu, R., et al. 2014. Characterization of extracellular matrix proteins during wound healing in the lamina propria of vocal fold in a canine model: A long-term and consecutive study. Acta Histochemica 116 (5): 730–735.

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. 18β-glycyrrhetinic acid ameliorates bleomycin-induced idiopathic pulmonary fibrosis via inhibiting TGF-β1/JAK2/STAT3 signaling axis;The Journal of Steroid Biochemistry and Molecular Biology;2024-10

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