Rehmannia glutinosa Suppresses Inflammatory Responses Elicited by Advanced Glycation End Products-Reference-Cited by-同舟云学术

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Rehmannia glutinosa Suppresses Inflammatory Responses Elicited by Advanced Glycation End Products

Published:2012-02-14 Issue:4 Volume:35 Page:1232-1241
ISSN:0360-3997
Container-title:Inflammation
language:en
Short-container-title:Inflammation

Author:

Baek Gui-Hyun,Jang Yong-Suk,Jeong Seung-Il,Cha Jaeho,Joo Myungsoo,Shin Sang-Woo,Ha Ki-Tae,Jeong Han-Sol

Publisher

Springer Science and Business Media LLC

Subject

Immunology,Immunology and Allergy

Link

http://link.springer.com/content/pdf/10.1007/s10753-012-9433-x.pdf

Reference57 articles.

1. Ahmed, N. 2005. Advanced glycation endproducts—role in pathology of diabetic complications. Diabetes Research and Clinical Practice 67: 3–21.

2. Bierhaus, A., M.A. Hofmann, R. Ziegler, and P.P. Nawroth. 1998. AGEs and their interaction with AGE-receptors in vascular disease and diabetes mellitus. I. The AGE concept. Cardiovascular Research 37: 586–600.

3. Singh, R., A. Barden, T. Mori, and L. Beilin. 2001. Advanced glycation end-products: a review. Diabetologia 44: 129–146.

4. Turk, Z., S. Ljubic, N. Turk, and B. Benko. 2001. Detection of autoantibodies against advanced glycation endproducts and AGE-immune complexes in serum of patients with diabetes mellitus. Clinica chimica acta; international journal of clinical chemistry 303: 105–115.

5. Yamamoto, Y., I. Kato, T. Doi, H. Yonekura, S. Ohashi, M. Takeuchi, et al. 2001. Development and prevention of advanced diabetic nephropathy in RAGE-overexpressing mice. The Journal of Clinical Investigation 108: 261–268.

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