Co-treatment with probucol does not improve lung pathology in hydroxypropyl-β-cyclodextrin-treated Npc1−/− mice
Author:
Funder
National Institutes of Health
Publisher
Springer Science and Business Media LLC
Subject
Genetics,General Medicine
Link
http://link.springer.com/article/10.1007/s13353-019-00487-x/fulltext.html
Reference22 articles.
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2. Chisolm GM III, Morel DW (1988) Lipoprotein oxidation and cytotoxicity: effect of probucol on streptozotocin-treated rats. Am J Cardiol 62:20B–26B
3. Condorelli S, Lombardi D, Gusmano R, Pisano L (1972) Pulmonary A-V differences of lipids and free fatty acids in relation to fasting an to high fat meals. Clin Chim Acta 38:a4a–aa46
4. Davidson CD, Ali NF, Micsenyi MC, Stephney G, Renault S, Dobrenis K, Ory DS, Vanier MT, Walkley SU (2009) Chronic cyclodextrin treatment of murine Niemann–Pick C disease ameliorates neuronal cholesterol and glycosphingolipid storage and disease progression. PLoS One 4(9):e6951. https://doi.org/10.1371/journal.pone.0006951
5. Deutsch G, Muralidhar A, Le E, Borbon IA, Erickson RP (2016) Extensive macrophage accumulation in young and old Niemann-Pick C1 model mice involves the alternative, M2, activation pathway and inhibition of macrophage apoptosis. Gene 578:242–250
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1. Intermolecular interactions between β-cyclodextrin and water;RSC Advances;2021
2. Construction of a sustainable/controlled-release nano-container of non-toxic corrosion inhibitors for the water-based siliconized film: Estimating the host-guest interactions/desorption of inclusion complexes of cerium acetylacetonate (CeA) with beta-cyclodextrin (β-CD) via detailed electronic/atomic-scale computer modeling and experimental methods;Journal of Hazardous Materials;2020-11
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