Effects of exercise preconditioning on NLRP3 and mitochondrial fission in isoproterenol-induced myocardial infarcted rats
Author:
Publisher
Springer Science and Business Media LLC
Subject
Pathology and Forensic Medicine,Anatomy
Link
https://link.springer.com/content/pdf/10.1007/s00580-022-03397-3.pdf
Reference51 articles.
1. An N, Gao Y, Si Z, Zhang H, Wang L et al (2019) Regulatory mechanisms of the NLRP3 inflammasome, a novel immune-inflammatory marker in cardiovascular diseases. Front Immunol 10:1592. https://doi.org/10.3389/fimmu.2019.01592
2. Archer SL (2013) Mitochondrial dynamics—mitochondrial fission and fusion in human diseases. N Engl J Med 369(23):2236–2251. https://doi.org/10.1056/nejmra1215233
3. Borges JP, Lessa MA (2015) Mechanisms involved in exercise-induced cardioprotection: a systematic review. Arq Bras Cardiol 105:71–81. https://doi.org/10.5935/abc.20150024
4. Cooper HA, Eguchi S (2018) Inhibition of mitochondrial fission as a novel therapeutic strategy to reduce mortality upon myocardial infarction. Clin Sci 132(20):2163–2167. https://doi.org/10.1042/cs20180671
5. Disatnik MH, Ferreira JC, Campos JC, Gomes KS, Dourado PM et al (2013) Acute inhibition of excessive mitochondrial fission after myocardial infarction prevents long-term cardiac dysfunction. J Am Heart Assoc 2(5):e000461. https://doi.org/10.1161/JAHA.113.000461
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1. NLRP3 inflammasome and pyroptosis in cardiovascular diseases and exercise intervention;Frontiers in Pharmacology;2024-04-12
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