Autoantibodies to heat shock proteins 60, 70, and 90 in patients with rheumatoid arthritis
Author:
Funder
Narodowe Centrum Nauki
Publisher
Springer Science and Business Media LLC
Subject
Cell Biology,Biochemistry
Link
https://link.springer.com/article/10.1007/s12192-018-0951-9/fulltext.html
Reference24 articles.
1. Chandawarkar RY, Wagh MS, Kovalchin JT, Srivastava P (2004) Immune modulation with high-dose heat-shock protein gp96: therapy of murine autoimmune diabetes and encephalomyelitis. Int Immunol 16:615–624
2. Guo Q, Wang Y, Xu D, Nossent J, Pavlos NJ, Xu J (2018) Rheumatoid arthritis: pathological mechanisms and modern pharmacologic therapies. Bone Res 6:15. https://doi.org/10.1038/s41413-018-0016-9
3. Kampinga HH, Hageman J, Vos MJ, Kubota H, Tanguay RM, Bruford EA, Cheetham ME, Chen B, Hightower LE (2009) Guidelines for the nomenclature of the human heat shock proteins. Cell Stress Chaperones 14:105–111. https://doi.org/10.1007/s12192-009-0106-0
4. Kasperkiewicz M, Tukaj S, Gembicki AJ, Silló P, Görög A, Zillikens D, Kárpáti S (2014) Evidence for a role of autoantibodies to heat shock protein 60, 70, and 90 in patients with dermatitis herpetiformis. Cell Stress Chaperones 9:837–843. https://doi.org/10.1007/s12192-014-0507-6
5. Koffeman EC, Genovese M, Amox D, Keogh E, Santana E, Matteson EL, Kavanaugh A, Molitor JA, Schiff MH, Posever JO, Bathon JM, Kivitz AJ, Samodal R, Belardi F, Dennehey C, van den Broek T, van Wijk F, Zhang X, Zieseniss P, le T, Prakken BA, Cutter GC, Albani S (2009) Epitope-specific immunotherapy of rheumatoid arthritis: clinical responsiveness occurs with immune deviation and relies on the expression of a cluster of molecules associated with T cell tolerance in a double-blind, placebo-controlled, pilot phase II trial. Arthritis Rheum 60:3207–3216. https://doi.org/10.1002/art.24916
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