Fatty acid-binding protein 4: a key regulator of ketoacidosis in new-onset type 1 diabetes
Author:
Funder
Israel Science Foundation
Publisher
Springer Science and Business Media LLC
Subject
Endocrinology, Diabetes and Metabolism,Internal Medicine
Link
https://link.springer.com/content/pdf/10.1007/s00125-021-05606-0.pdf
Reference31 articles.
1. Furuhashi M, Hotamisligil GS (2008) Fatty acid-binding proteins: role in metabolic diseases and potential as drug targets. Nat Rev Drug Discov 7(6):489–503. https://doi.org/10.1038/nrd2589
2. Hotamisligil GS, Bernlohr DA (2015) Metabolic functions of FABPs - mechanisms and therapeutic implications. Nat Rev Endocrinol 11:592–605. https://doi.org/10.1038/nrendo.2015.122
3. Ertunc ME, Sikkeland J, Fenaroli F et al (2015) Secretion of fatty acid binding protein aP2 from adipocytes through a nonclassical pathway in response to adipocyte lipase activity. J Lipid Res 56(2):423–434. https://doi.org/10.1194/jlr.M055798
4. Mita T, Furuhashi M, Hiramitsu S et al (2015) FABP4 is secreted from adipocytes by adenyl cyclase-PKA- and guanylyl cyclase-PKG-dependent lipolytic mechanisms. Obesity (Silver Spring) 23(2):359–367. https://doi.org/10.1002/oby.20954
5. Syamsunarno MRAA, Iso T, Hanaoka H et al (2013) A critical role of fatty acid binding protein 4 and 5 (FABP4/5) in the systemic response to fasting. PLoS One 8(11):e79386. https://doi.org/10.1371/journal.pone.0079386
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