Complete mid-portion rupture of the rat achilles tendon leads to remote and time-mismatched changes in uninjured regions
Author:
Funder
Sahlgrenska Universitetssjukhuset
Publisher
Springer Science and Business Media LLC
Subject
Orthopedics and Sports Medicine,Surgery
Link
https://link.springer.com/content/pdf/10.1007/s00167-020-06239-3.pdf
Reference35 articles.
1. Abraham T, Fong G, Scott A (2011) Second harmonic generation analysis of early Achilles tendinosis in response to in vivo mechanical loading. BMC Musculoskelet Disord 12:26
2. Aikawa E, Whittaker P, Farber M et al (2006) Human semilunar cardiac valve remodeling by activated cells from fetus to adult: implications for postnatal adaptation, pathology, and tissue engineering. Circulation 113:1344–1352
3. Asai S, Otsuru S, Candela ME, Enomoto-Iwamoto M et al (2014) Tendon progenitor cells in injured tendons have strong chondrogenic potential: the CD105-negative subpopulation induces chondrogenic degeneration. Stem Cells 32:3266–3277
4. Ateschrang A, Gratzer C, Weise K (2008) Incidence and effect of calcifications after open-augmented Achilles tendon repair. Arch Orthop Trauma Surg 128:1087–1092
5. Bring DK, Paulson K, Renstrom P et al (2012) Residual substance P levels after capsaicin treatment correlate with tendon repair. Wound Repair Regen 20:50–60
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