Sexually dimorphic distribution of Prokr2 neurons revealed by the Prokr2-Cre mouse model
Author:
Funder
National Institute of Child Health and Human Development
Publisher
Springer Science and Business Media LLC
Subject
Histology,General Neuroscience,Anatomy
Link
http://link.springer.com/article/10.1007/s00429-017-1456-5/fulltext.html
Reference90 articles.
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2. Abreu AP, Kaiser UB, Latronico AC (2010) The role of prokineticins in the pathogenesis of hypogonadotropic hypogonadism. Neuroendocrinology 91:283–290. doi: 10.1159/000308880
3. Abreu AP, Noel SD, Xu S, Carroll RS, Latronico AC, Kaiser UB (2012) Evidence of the importance of the first intracellular loop of prokineticin receptor 2 in receptor function. Mol Endocrinol 26:1417–1427. doi: 10.1210/me.2012-1102
4. Allen SJ, Garcia-Galiano D, Borges BC, Burger LL, Boehm U, Elias CF (2016) Leptin receptor null mice with re-expression of LepR in GnRH-R expressing cells display elevated FSH levels but remain in a prepubertal state Am J Physiol Regul Integr Comp Physiol 00529:02015. doi: 10.1152/ajpregu.00529.2015
5. Asakura Y et al (2015) Combined pituitary hormone deficiency with unique pituitary dysplasia and morning glory syndrome related to a heterozygous PROKR2 mutation. Clin Pediatr Endocrinol 24:27–32. doi: 10.1297/cpe.24.27
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