Pathogenese der akuten Pankreatitis
Author:
Publisher
Springer Science and Business Media LLC
Subject
Internal Medicine
Link
https://link.springer.com/content/pdf/10.1007/s00108-021-01158-y.pdf
Reference29 articles.
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2. Algül H, Treiber M, Lesina M et al (2007) Pancreas-specific RelA/p65 truncation increases susceptibility of acini to inflammation-associated cell death following cerulein pancreatitis. J Clin Invest 117:1490–1501. https://doi.org/10.1172/JCI29882
3. Bertheloot D, Latz E, Franklin BS (2021) Necroptosis, pyroptosis and apoptosis: an intricate game of cell death. Cell Mol Immunol 18:1106–1121. https://doi.org/10.1038/s41423-020-00630-3
4. Biczo G, Vegh ET, Shalbueva N et al (2018) Mitochondrial dysfunction, through impaired autophagy, leads to endoplasmic reticulum stress, deregulated lipid metabolism, and pancreatitis in animal models. Gastroenterology 154:689–703. https://doi.org/10.1053/j.gastro.2017.10.012
5. Dawra R, Sah RP, Dudeja V et al (2011) Intra-acinar trypsinogen activation mediates early stages of pancreatic injury but not inflammation in mice with acute pancreatitis. Gastroenterology 141:2210–2217.e2. https://doi.org/10.1053/j.gastro.2011.08.033
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